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Section 1: Introduction, Section 2: Physical Features, Section 3: Biological Resources, Section 4: Social/Economic, Section 5: Important Resources, Section 6: Watershed Classification, Section 7: Watershed Management, Section 8: Watershed Planning, Appendix A: Water Quality Data and Assessments, Appendix B: Selected References, Appendix C: RUSLE, Appendix D: AGWA

In December 2007 I began an internship with the Tennessee Williams Scholars' Conference/Tennessee Williams Annual Review. The Conference and Review are co-produced by Williams expert and professor of English at Middle Tennessee State University Dr. Robert Bray and the publications department of The Historic New Orleans Collection. The majority of my internship took place from my home and at The Collection, where I also currently serve full-time as an editor on book projects and the institution's quarterly magazine. The following report provides an overview of my internship, my analysis of the operation of the Conference/Review, and my recommendations for the organization developed over the course of my tenure, which will conclude in December 2008.

Orientador: Luciana Pinato / Banca: Renata Frazão / Banca: Célia Maria Giacheti / Resumo: A síndrome de Williams-Beuren (SWB), causada por uma microdeleção na região cromossômica 7q.11.23, apresenta como fenótipo: características faciais típicas, baixa estatura, anormalidades cardiovasculares e do tecido conjuntivo, perfil cognitivo e personalidade ímpar, além de deficiência intelectual em diferentes graus. Recentes estudos relatam alta prevalência de distúrbios do sono nesta população os quais podem indicar uma possível disfunção em sua ritmicidade biológica, agravando seus problemas comportamentais e de aprendizagem. Apesar das consequências negativas dos distúrbios do sono sobre o desempenho cognitivo e aspectos comportamentais, poucos estudos exploraram esta questão e ainda não está claro se o padrão de sono-vigília apresenta correlação com o padrão comportamental na SWB. O sono é um fenômeno biológico controlado por uma complexa rede neural composta por vias de sinalização e processos regulatórios nos níveis molecular, celular e organísmico. Neste cenário, a melatonina, hormônio produzido pela glândula pineal na fase de escuro, apresenta comprovada importância para o início, manutenção e qualidade do sono, sendo que, alterações em sua síntese estão muitas vezes associadas às causas dos distúrbios de sono. Distúrbios do sono, por sua vez, podem estar relacionados com alterações de memória, que compõem o fenótipo da SWB. Assim sendo, este estudo teve o intuito de caracterizar o padrão de 10 sono-vigília, de comportamento e avaliar os níveis de atenção e memória na SWB, e correlacioná-los aos níveis de melatonina em SWB. O grupo pesquisa (GP) foi composto por 15 crianças de 6 a 17 anos, com SWB, e o grupo controle (GC) composto por 15 crianças pareadas ao GP por idade, com ausência de histórico para doenças genéticas, neurológicas ou alterações de desenvolvimento cognitivo e motor. Foram utilizados para a coleta de dados a Escala de... / Abstract: The Williams-Beuren syndrome (WBS), caused by a microdelection in the chromosomal region 7q.11.23, shows a phenotype as typical facial features, short stature, cardiovascular abnormalities and connective tissue disorders, cognitive profile and unique personality, besides intellectual disability in different degrees. Recent studies reported high prevalence of sleep problems in this population which may indicate a possible biological rhythm disorder in these individuals, worsening their behavior and learning problems. Despite the negative consequences of sleep disorders on cognitive performance and behavioral aspects, few studies have explored this issue and it is unclear whether the sleep-wake pattern is correlated with behavioral patterns in WBS. Sleep is a biological phenomenon controlled by a complex neural network consisting of signaling pathways and regulatory processes in molecular, cellular and organismic levels. In this scenario, melatonin, a hormone produced by the pineal gland during the dark, has been showed important for the initiation, maintenance and quality of sleep, and that changes in its synthesis are often associated with the causes of sleep disorders. Sleep disorders can be related with memory problems, component of the WBS phenotype. Therefore this study is aimed at characterizing the pattern of sleep-wake behavior, assess levels of attention and memory, and correlate them to melatonin levels in WBS. The study group (GP) consisted of 15 children 6-17 years old with WBS, and the control group (GC) composed of 15 children matched for age, with no history to genetic disorders, or neurological changes in cognitive and motor development. For data collection, were used the Sleep Disturbance Scale for Children (EDSC), the Child Behavior Checklist for ages 6-18 (CBCL) parents version, WISC-III and WAIS-III for the characterization of cognitive abilities, visual sequential memory subtest of the brazilian 13 version of the Illinois... / Mestre

Thesis (M.A. (Drama))--Rhodes University, 2003. / "A thesis sumbitted in partial fulfiment of the requirements for the degree of Master of Arts."

Histories o f childhood and youth have generally focused upon social policy toward young people. This dissertation chronicles the actual experiences of youth growing up in and around Williams Lake in the Cariboo-Chilcotin region of British Columbia, a "western" community surrounded by open spaces, ranches, and Aboriginal reservations. Williams Lake underwent economic, demographic, spatial as well as social transformation in the first three decades following the Second World War. Forty-three oral interviews with two sets o f subjects who were adolescents in the study area furnished the bulk of the primary evidence. Most of the first "generation" were bom in the Great Depression and were teens sometime between 1945 and 1955. The second generation are "baby-boomers" bom between 1947 and 1962 who were teens between 1965 and 1975. This joint narrative details select aspects o f their lives at school, at paid and unpaid labour, with friends, and at leisure. It suggests changes and continuities in the experience of local youth between 1945 and 1975. First generation non-Aboriginal subjects grew up with a somewhat coherent peer group albeit with relatively little physical and social contact with Aboriginal youth. Gendered domestic labour around home and property honed work skills and dispositions from an early age. The emergence of local sawmills greatly expanded work options for males but not females. Males also enjoyed comparatively more spatial and temporal freedom throughout their youth. Second generation subjects grew up in a context of greater urbanization and access to mass culture. The merger of regional youth in the high school along with natural population growth, demographic change including the enrollment of first Aboriginal and then Indo-Canadian youth encouraged factions as well as cultural gulfs among youth in the school and community. Their leisure was comparatively less divided, at least on the basis of gender, as many non-Aboriginal parents eased traditional restrictions upon daughters. With notable exceptions this generation contributed less labour to their household and directed part- and full-time earnings into satisfying their own personal interests. The author suggests the pattern of youths' recreational use of hinterlands during the period reflects common practice in many Canadian communities located in similar rural and isolated settings. He illustrates how factors such as family affluence and circumstances, gender, "race" and ethnicity continued to mediate the experience of growing up in this post-war period. He concludes many more local accounts of the experiences of youth are needed before any attempt is made at an inclusive national historical synthesis of growing up in Canada after the Second World War.

Thesis (M.A.)--Georgia State University, 2009. / Title from title page (Digital Archive@GSU, viewed July 16, 2010) Matthew Roudane, committee chair; Pearl McHaney, Wayne Erickson, committee members. Includes bibliographical references (p. 85-88).

Thesis (Ph. D.)--University of Wisconsin, Madison, 1942. / Includes bibliographical references (p. 342-346).

Thesis (M.A.)--Bowling Green State University, 2006. / Document formatted into pages; contains v, 66 p. Includes bibliographical references.

Made available in DSpace on 2014-12-02T11:16:35Z (GMT). No. of bitstreams: 0 Previous issue date: 2014Bitstream added on 2014-12-02T11:21:12Z : No. of bitstreams: 1 000797359.pdf: 541624 bytes, checksum: 9f5615e2b4a01833b695a6dfe8afd56b (MD5) / Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES) / A síndrome de Williams-Beuren (SWB), causada por uma microdeleção na região cromossômica 7q.11.23, apresenta como fenótipo: características faciais típicas, baixa estatura, anormalidades cardiovasculares e do tecido conjuntivo, perfil cognitivo e personalidade ímpar, além de deficiência intelectual em diferentes graus. Recentes estudos relatam alta prevalência de distúrbios do sono nesta população os quais podem indicar uma possível disfunção em sua ritmicidade biológica, agravando seus problemas comportamentais e de aprendizagem. Apesar das consequências negativas dos distúrbios do sono sobre o desempenho cognitivo e aspectos comportamentais, poucos estudos exploraram esta questão e ainda não está claro se o padrão de sono-vigília apresenta correlação com o padrão comportamental na SWB. O sono é um fenômeno biológico controlado por uma complexa rede neural composta por vias de sinalização e processos regulatórios nos níveis molecular, celular e organísmico. Neste cenário, a melatonina, hormônio produzido pela glândula pineal na fase de escuro, apresenta comprovada importância para o início, manutenção e qualidade do sono, sendo que, alterações em sua síntese estão muitas vezes associadas às causas dos distúrbios de sono. Distúrbios do sono, por sua vez, podem estar relacionados com alterações de memória, que compõem o fenótipo da SWB. Assim sendo, este estudo teve o intuito de caracterizar o padrão de 10 sono-vigília, de comportamento e avaliar os níveis de atenção e memória na SWB, e correlacioná-los aos níveis de melatonina em SWB. O grupo pesquisa (GP) foi composto por 15 crianças de 6 a 17 anos, com SWB, e o grupo controle (GC) composto por 15 crianças pareadas ao GP por idade, com ausência de histórico para doenças genéticas, neurológicas ou alterações de desenvolvimento cognitivo e motor. Foram utilizados para a coleta de dados a Escala de... / The Williams-Beuren syndrome (WBS), caused by a microdelection in the chromosomal region 7q.11.23, shows a phenotype as typical facial features, short stature, cardiovascular abnormalities and connective tissue disorders, cognitive profile and unique personality, besides intellectual disability in different degrees. Recent studies reported high prevalence of sleep problems in this population which may indicate a possible biological rhythm disorder in these individuals, worsening their behavior and learning problems. Despite the negative consequences of sleep disorders on cognitive performance and behavioral aspects, few studies have explored this issue and it is unclear whether the sleep-wake pattern is correlated with behavioral patterns in WBS. Sleep is a biological phenomenon controlled by a complex neural network consisting of signaling pathways and regulatory processes in molecular, cellular and organismic levels. In this scenario, melatonin, a hormone produced by the pineal gland during the dark, has been showed important for the initiation, maintenance and quality of sleep, and that changes in its synthesis are often associated with the causes of sleep disorders. Sleep disorders can be related with memory problems, component of the WBS phenotype. Therefore this study is aimed at characterizing the pattern of sleep-wake behavior, assess levels of attention and memory, and correlate them to melatonin levels in WBS. The study group (GP) consisted of 15 children 6-17 years old with WBS, and the control group (GC) composed of 15 children matched for age, with no history to genetic disorders, or neurological changes in cognitive and motor development. For data collection, were used the Sleep Disturbance Scale for Children (EDSC), the Child Behavior Checklist for ages 6-18 (CBCL) parents version, WISC-III and WAIS-III for the characterization of cognitive abilities, visual sequential memory subtest of the brazilian 13 version of the Illinois...