Several cases of single repeat offenders in urban space have raised public concern in Sweden during recent decades. Few studies have been conducted on consequences of the kind of ‘hostage situations’ that emerge when one individual offender causes fear and affects a larger group of people in a specific place. The concern of this thesis is to examine consequences of the Haga Man phenomenon: the case of a serial rapist operating between 1998 and 2006 in Umeå, a medium-sized Swedish city. This thesis explores some of the ways not only women but also men in Umeå responded to this specific situation, the threat from a single repeat offender, and how fear of crime and changing public crime discourses influenced gendered power relations. The thesis examines different aspects of fear and safety in public space, such as the views of those who are fearful; of those who are feared; perceptions of both women’s and men’s bodies; their emotions and experiences in relation to fear of violence in public spaces; and the significance of space and place for our understanding of fear. The empirical data of this thesis consist of in-depth interviews with a total of 47 women and men in Umeå. The thesis is based on four empirical studies. The first (Paper I) sought to identify similarities and differences across narratives in terms of the major components of young people’s talk about fear.  In their stories women positioned themselves as fearful and in need of protection, while men in their stories positioned themselves as fearless protectors. Men and women reproduced ways of speaking considered appropriate to their gender, thus performing masculinity and femininity through their talk. Paper II, examines consequences of the Haga Man phenomenon on constructions of white masculinities. Three masculine positions; the dangerous stranger, the suspect and the protector were identified. These three constructions of masculinity were not clear-cut or ‘belonging’ to specific men – several of the interviewees articulated various forms of masculinities but stressed them in different ways depending on, for instance, age and/or ethnicity/race. Paper III, focuses on changing perceptions and representations of female and male bodies, and illustrates how a change took place; from a focus on how women should conduct themselves to be safe, towards men’s bodily behaviour in order to present themselves in non-threatening ways. In Paper IV, women’s fear of violence is discussed in relation to Swedish gender equality discourses and contextual constructions of femininity. The results show the difficulties of claiming the official position of a gender-equal femininity. Several female respondents expressed an ambivalent attitude about their own fear; they felt afraid, but also felt that as (equal) women they should be able to do what they wanted, whenever they wanted.  Result from this thesis shows that this situation produced a shared approach to fear for women of different ages, classes and ethnicities in Umeå. The similarity in the women’s responses to the threat from the Haga Man is as an expression of a normative femininity. The male respondents did on the other hand express complex emotional positions as they talked about their own fears, women’s fear of unknown men and how they felt they were under suspicion and compared to the perpetrator. As this thesis provides an understanding of how men and women responded and reacted to the threat from the Haga man, it contributes to a better understanding of how fear of violence affects people in their everyday lives.

The present thesis examined extinction of conditioned fear in the developing rat. In the adult rat, the hippocampus is thought to be important for the context-specificity of extinction. Because the hippocampus is a late-maturing structure, it was hypothesised that context-modulation of extinction may be different across development. The first series of experiments investigated reinstatement of extinguished fear in the developing rat (Chapter 2). The results showed that P24 rats exhibited context-specific reinstatement. On the other hand, P17 rats did not exhibit reinstatement of extinguished fear following a US reminder treatment. The failure to see reinstatement in P17 rats was not due to the reminder treatment being ineffective in these rats because the same treatment alleviated spontaneous forgetting in rat this age. The second series of experiments then examined the renewal effect and GABAergic involvement in extinction in P24 and P17 rats (Chapter 3). It was observed that P24 rats displayed renewal whereas P17 rats did not. Also, pre-test injection of FG7142 recovered extinguished fear in P24 rats but not in P17 rats, even across a range of doses. This failure to see any FG7142 effect on extinction in P17 rats was not due to the lack of responsiveness to this drug in these rats because FG7142 was found to be effective in alleviating spontaneous forgetting in rats this age. The third series of experiments then examined the effect of temporary inactivation of the amygdala on extinction and re-extinction in the developing rat (Chapter 4). It was observed that extinction retention is impaired in both P24 and P17 rats if the amygdala is inactivated during extinction training. Interestingly, when a CS that had been previously extinguished and then re-trained was re-extinguished, re-extinction was amygdala-independent if initial extinction occurred at 24 days of age but amygdala-dependent if initial extinction occurred at 17 days of age. That is, amygdala involvement in re-extinction was dissociated across development. Taken together, these experiments provide strong evidence for fundamental differences in mechanisms underlying fear extinction across development. The implications of the findings were discussed in light of the theoretical and neural models of extinction.

N-methyl-D-asparate (NMDA) receptors located within the basolateral complex of the amygdala are required for the consolidation and expression of Pavlovian conditioned fear. The events downstream of receptor activation that mediate these processes are not well defined. An intermediate step that may be of significance is the synthesis of the gas nitric oxide (NO). Nitric oxide is synthesised as a result of NMDA receptor activation and acts as an unconventional neurotransmitter freely diffusing across cell membranes interacting with its targets in a non-synaptic manner. The targets of NO include cellular components that play significant roles during the consolidation of conditioned fear and the neurotransmission associated with its expression. This implies that NO may be an important intermediary of NMDA receptor activation and both these processes. The current study sought to examine this possibility using fear potentiated startle to examine the expression of learned fear. Three experiments were conducted, fifty rats received intra-BSC microinfusions of the global nitric oxide synthase inhibitor L-NAME either prior to fear conditioning, fear testing, or examination of the shock sensitization of the acoustic startle affect. The results indicated that NO was indeed required for both the consolidation and expression of learned fear, whereas it was not required for shock enhanced startle responding. This study provides new information about the sub-cellular basis of conditioned fear, and highlights the pivotal role played by NO in processes associated with conditioned fear.

The ventral tegmental area (VTA) and its dopaminergic (DA) mesocorticolimbic projections are thought to be essential in the brain’s reward neurocircuitry. In humans and animal experimental subjects, mild electrical VTA stimulation increases dopamine levels and can induce euphoria. Paradoxically, aversive stimuli activate VTA neurons and forebrain DA activity, and excessive electrical stimulation of the VTA exaggerates fearfulness. Research suggests that experimental manipulation of either the amygdala or the VTA has similar effects on the acquisition and expression of Pavlovian conditioned fear. Recently it was demonstrated that electrical stimulation of the amygdala produced fear extinction deficits in rats. Fear extinction involves the progressive dissipation of conditioned fear responses by repeated non-reinforced exposure to a conditioned stimulus (CS). Maladaptive states of fear in fear-related anxiety disorders, such as post-traumatic stress disorders (PTSD) or specific phobias are thought to reflect fear extinction learning deficits. The primary purpose of the present study was to examine the effects of intra-VTA stimulation on fear extinction learning. Using fear-potentiated startle as a behavioural index of conditioned fear, it was found that 120 VTA stimulations paired or unpaired with non-reinforced CS presentations impaired the extinction of conditioned fear. This effect was not apparent in rats that received electrical stimulation of the substantia nigra (SN), suggesting that not all midbrain regions respond similarly. Electrical stimulation parameters did not have aversive affects because rats failed to show fear conditioning when electrical VTA stimulation was used as the unconditioned stimulus. Also, VTA stimulation did not alter conditioned fear expression in non-extinguished animals. Based on the results it is suggested that VTA activation disinhibited conditioned fear responding. Therefore, VTA neuronal excitation by aversive stimuli may play a role in fear-related anxiety disorders thought to reflect extinction learning deficits.

Thesis (Ph. D.)--West Virginia University, 2009. / Title from document title page. Document formatted into pages; contains ix, 192 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 171-190).

The automobile is a cultural artifact embedded in our lives and imbued with meaning. Autonomous vehicle technology stands to alter not just the way we drive or whether we drive, it also has the power to fundamentally change the way we live. The development of driverless cars enables the examination of the complex relationships that individuals have with the automobile and reveals the fears associated with this technological change.

Fear of failure (FF) har negativ påverkan på individers motivation, val av arbete, prestationsförmåga och allmänt välbefinnande och kan uppstå när människor skall ta olika beslut. Vår studie avsåg att undersöka om fear of failure kan förklaras av personlighet och om det fanns andra påverkansfaktorer, så som socioekonomisk status, ålder och kön. En kvantitativ metod användes och datainsamlingen skedde via en webbaserad enkät innehållande 62 frågor. Deltagarna i studien (N = 153), bestod av 115 kvinnor (75%) och 38 män (25%). IPIP-30 användes för att mäta personlighet och Performance Failure Appraisal Inventory (PFAI) användes för att mäta fear of failure. Resultatet visade i regressionsanalys ett mycket starkt statistiskt signifikant samband (r = .69) mellan fear of failure och personlighetsfaktorn neuroticism, samt att det kunde förklara ≈ 53% av variansen inom fear of failure. Resultatet var förvånansvärt starkt och tydligt och visade på ett övertygande sätt att personlighet förklarade en stor del av innehållet i det begrepp som kallas "fear of failure". Vår slutsats är därför att fear of failure som begrepp, tillsammans med tillhörande mätinstrument (PFAI-skalan), blir överflödigt och kan istället ersättas av personlighetsfaktorerna i femfaktormodellen. / Fear of failure (FF) has a negative impact on individuals' motivation, choice of occupation, ability to work and general well-being and can occur when people make different decisions. Our study was designed to investigate whether fear of failure can be explained by personality and whether there were other factors of influence such as socioeconomic status, age and sex. A quantitative method was used, and data collection was conducted via a web-based survey containing 62 questions. The participants in the study (N = 153) consisted of 115 women (75%) and 38 men (25%). IPIP-30 was used to measure personality and the Performance Failure Appraisal Inventory (PFAI) was used to measure fear of failure. The result showed in regression analysis a very strong statistically significant relationship (r = .69) between fear of failure and the personality factor neuroticism, and that it could explain ≈ 53% of the variance in fear of failure. The result was surprisingly strong and clear and showed convincingly that personality explained a large part of the content of the term called "fear of failure". Our conclusion is therefore that fear of failure as a concept, together with the associated measuring instrument (PFAI scale) becomes superfluous and can instead be replaced by the personality in the five-factor model.

A amígdala participa dos processos de aprendizagem e memória de natureza emocional, incluindo os comportamentos aversivos. Essa estrutura compreende vários núcleos que estabelecem diferentes conexões com outras estruturas do sistema nervoso. Seu núcleo basal (BA) é um dos principais alvos amigdalares de informações processadas pelo hipocampo. Evidências apontam que o hipocampo seria responsável pela aprendizagem contextual, construindo uma representação integrada dos diferentes estímulos do ambiente numa representação única, denominada representação configuracional do contexto, que inclui também representações do espaço. Congruente com essa hodologia, lesões seletivas do BA resultam em prejuízos comportamentais similares aos encontrados após lesão hipocampal. Por exemplo, ratos com lesão no BA exibem deficiências na tarefa de medo condicionado ao contexto, mas não ao som, indicando que essa região está envolvida no processo de condicionamento aversivo contextual. Porém, não está claro se esse prejuízo decorre da participação do BA na aquisição e/ou evocação e expressão do medo contextual. Os objetivos do presente estudo incluíram avaliar, por meio da inativação reversível do BA, (1) se ele é necessário na aquisição do condicionamento aversivo ao som e/ou ao contexto e (2) qual a natureza da sua participação no condicionamento aversivo ao contexto, isto é, se é necessário para a construção do contexto, para sua associação com o estímulo aversivo e/ou para a evocação da memória e expressão das respostas condicionadas. Num primeiro experimento, muscimol foi infundido no BA antes do treinamento na tarefa de condicionamento aversivo concorrente ao som e ao contexto, e os testes de medo condicionado ao som e ao contexto foram realizados separadamente, na ausência de muscimol. Como esperado, os resultados revelaram prejuízo de desempenho na tarefa de medo condicionado ao contexto, mas não na tarefa de medo condicionado ao som. Num segundo experimento, muscimol foi infundido, em grupos independentes de animais, antes de cada fase da variante do condicionamento aversivo ao contexto que envolve facilitação pela pré-exposição ao contexto, a qual permite distinguir entre a construção configuracional do contexto (fase 1), sua associação com o estímulo aversivo (fase 2) e posterior evocação e expressão do medo condicionado (fase 3). Resultados mostraram que somente os ratos que receberam muscimol antes da fase 2, mas não os que receberam muscimol antes das fases 1 e 3, apresentaram prejuízo de desempenho na tarefa de medo condicionado ao contexto. No conjunto, esses resultados indicam que o BA participa do condicionamento aversivo ao contexto sendo imprescindível no processo de associação da representação configuracional do contexto com o estímulo aversivo, mas não nos processos de construção da representação sobre o contexto e nem de evocação da memória e expressão das respostas condicionadas de medo / The amygdala is involved in emotional learning and memory, including fear conditioning. This brain structure includes several nuclei with distinct hodology. The basal nucleus (BA) receives processed information from the hippocampal formation. Evidence indicates that the hippocampus integrates environmental stimuli in a single representation thus rendering it involved in contextual (including spatial) learning and memory. Congruent with this hodological evidence, selective damage to the BA results in behavioral impairments similar to those found after hippocampal damage. For instance, rats with BA damage exhibit performance impairments in contextual, but not auditory, fear conditioning tasks. However, it is not clear to which extent this disruption is related to the BA involvement in either acquisition of contextual fear or retrieval of memory and fear expression, or both. This study aimed at investigating, by way of reversible inactivation of the BA, (1) its involvement in acquisition of auditory and contextual fear conditioning, and (2) the nature of its participation in contextual fear conditioning, that is, if it is necessary for building a representation about the context, for associating the context with the aversive stimulus and/or for memory retrieval and expression of contextual fear conditioning. In the first experiment, muscimol was infused into the BA before training in a concurrent auditory and contextual fear conditioning task, and testing for auditory and contextual fear conditioning was run separately in the absence of muscimol. As expected, results revealed disruption of performance in the contextual, but not in the auditory, conditioning task. In the second experiment, muscimol was infused into the BA, in independent groups of animals, before each of the three phases of a contextual fear conditioning variant that involves context pre-exposure facilitation, thus allowing to evaluate if functional reversible inactivation of the BA interfered with (1) building an integrated representation of the environment (the context), (2) its association with the aversive stimulus, and/or (3) memory retrieval and expression of fear conditioning. Results showed that muscimol infusion into BA before phase 2, but not phases 1 and 3, impaired performance in the contextual fear conditioning task. Together, these results indicate that a functional BA is required for acquisition of contextual fear conditioning in order to establish an association between the context and the aversive stimulus, but not for building a context neither for memory retrieval and expression of fear conditioned responses

Master of Arts / Department of Sociology, Anthropology, and Social Work / L. Susan Williams / Studies on fear of crime demonstrate that fear of rape controls women’s lives by altering emotions and behavior, though how women construct rape discourse through social networks has not been examined. Further, studies tend to dismiss stranger rape because of its rarity compared to acquaintance rape, but this study argues that research must begin where women are. This study looks to women’s voices to articulate how they talk about fear of rape; specifically, it examines responses to a serial rapist at work in a college town. Framed by feminist methodology, this study establishes the influence of fear on women’s lives and the role of women’s social networks in disseminating information, constructing strategies, and changing behavior as it relates to a local serial rapist. The study utilizes a multi-method approach to quantify levels of fear in the community and to document qualitatively women’s responses to knowledge about the serial rapist. Two surveys, content analysis of local newspapers, and interviews support this research. In particular, group interviews conducted in two environments – campus face-to-face groups and online virtual groups – provide opportunities for young women to voice concerns and report behavioral changes related to the serial rapes. The research demonstrates that women are concerned about insufficient information from formal sources and want more accurate reporting. Women depend heavily on informal networks for information, but it is often incomplete and/or inaccurate and may actually intensify fear. As documented in earlier research, women focus on stranger rape to the neglect of the more common acquaintance rape and tend to strategize in individual terms rather than recognize structural issues. A major finding of this research is that young women actually perceive a change in their own identity as they try to manage fear of rape. However, women’s social networks and, in particular, the increasingly popular online networks, provide a forum from which to try out strategies, build collective discourse, and, in turn, develop greater group consciousness among young women. From the experiences of women in this study, several policy implications are offered for managing fear, including education about the more likely threat of acquaintance rape.

The mechanisms underlying cannabinoid impairment of fear memory is not clear. This study investigated the effects of the synthetic cannabinoid HU210 and the endocannabinoid hydrolysis inhibitor JZL 195 on fear memory following contextual fear conditioning (CFC; an animal model of fear). The long-term depression (LTD)-blocking peptide Tat-GluR2 was utilized to investigate whether the expression of cannabinoid-induced LTD (CB-LTD) is required for the cannabinoid impairment of acquisition and consolidation of contextual fear memory. HU210 reduced freezing throughout the test phase of the acquisition protocol, which was not affected by pre-administration of Tat-GluR2. High and moderate doses of HU210 reduced freezing during the first and last half, respectively, of the test phase of the consolidation protocol, which was prevented by pre-treatment with Tat-GluR2. HU210 did not affect freezing during the test phase of the retrieval protocol. Thus, these results suggest that HU210 impairs acquisition and consolidation, but not retrieval of contextual fear memory, and that in vivo CB-LTD expression is required for HU210 impairment of the consolidation, but not acquisition, of contextual fear memory. We also observed that HU210 and JZL 195 do not facilitate the acquisition of contextual fear memory extinction.