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Introduktion: Plyometrisk träning är en högintensiv träningsform för att förbättra atletisk prestation som exempelvis explosiv styrka och effektutveckling. Plyometrisk träning i vatten är däremot mindre undersökt men tidigare forskning tyder på att det verkar ha liknande effekter som landbaserad plyometrisk träning. Syfte: Litteraturundersökningens syfte är att undersöka om det finns en effekt av plyometrisk träning i vatten på hopphöjd, muskelstyrka och muskelömhet. Ytterligare ett syfte är att jämföra effekterna mellan plyometrisk träning i vatten med landbaserad plyometrisk träning på hopphöjd, muskelstyrka och muskelömhet. Metod: En systematisk sökning genomfördes i de vetenskapliga databaserna Pubmed och Web of Science. Utöver detta genomsöktes samtliga referenser på de artiklar som uppfyllde inklusionskriterierna. Resultat: Litteraturundersökningen visade att plyometrisk träning i vatten verkar ha en effekt på hopphöjd. Muskelstyrka och muskelömhet visade inget tydligt utfall. Plyometrisk träning i vatten verkar ha samma effekt på hopphöjd och muskelstyrka som landbaserad plyometrisk träning. Vattenbaserad plyometrisk träning verkar ge mindre muskelömhet jämfört med landbaserad plyometrisk träning. Konklusion: Det behövs mer forskning i området för att fastställa vilka faktorer som gör att vattenbaserad plyometrisk träning är lika effektiv som landbaserad träning för att förbättra hopphöjd. Det behövs mer forskning på plyometrisk träning i vatten associerad till muskelstyrka och muskelömhet för att få en tydligare bild av effekterna / Introduction: Effects of plyometric training to improve athletic performance such as explosive strength and power development are well-documented. However, less is known about the effects of aquatic plyometric training. Previous research suggests that it appears to have similar effects as land-based plyometric training. Purpose: The purpose of the literature review was to investigate if there is an effect of aquatic plyometric training on jump height, muscle strength and muscle soreness. Another aim was to compare the effects of plyometric training in water with land-based plyometric training on jump height, muscle strength and muscle soreness. Method: A systematic literature search was conducted in the scientific databases Pubmed and Web of Science. In addition, all references were manually searched on the articles that met the inclusion criteria. Results: The literature review shows that aquatic plyometric training seems to have an effect on jump height. Muscle strength and muscle soreness showed no clear outcome. Aquatic plyometric training seems to have the same effect on jump height and muscle strength as land-based plyometric training. Aquatic plyometric training seems to give less muscle soreness compared to land-based plyometric training. Conclusion: More research is necessary to determine which factors make aquatic plyometric training as effective as land-based plyometric training to improve jump height. More research is needed on aquatic plyometric training associated with muscle strength and muscle soreness to get a better understanding of the effects.

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These original data for this research were documented in the clinical diary records of an army psychiatrist on deployment in Vietnam during 1969–70. This study is unique due to the original battlefield diagnosis data used for foundation comparison analysis and longitudinal retrospective case control paired measurement. In battlefield psychiatric assessment diagnostic data recorded in Vietnam during 1969–70 of 119 Australian military servicemen (Experimental group) who presented battlefield trauma exposure reactions were examined. The research case controls (Control group) are 275 Australian Vietnam veterans selected from data at the Australian War Memorial Research Centre. Case control identified participants did not present with medical symptoms in 1969-70 and presented the same demographic profile as the Experimental group population. This research examined whether initial psychiatric illnesses initiated by battlefield trauma exposure in 1969-70 by a cohort of Vietnam veterans would have long term pernicious effects on their physical and psychological health, relationships and employment status. This research compared, PTSD, delayed onset PTSD, severity of combat exposure and depressive symptoms, quality of dyads, general health and quality of life. The analysis of specific demographic variables determined the means, standard deviations, and medians for those continuous variables for both groups from 1969-70 (n=394) and 2006-07 (n=97). The 2006-07 Experimental group (n=21) represents 17.65% and the Control group (n=76) represents 28.15% of the original groups selected and matched from 1969-70 data. These participants completed a battery of psychometric questionnaires and a follow up telephone interview. Demographic variables were evaluated for inclusion as covariates. These demographic variables were correlated with combat exposure and the presentation of PTSD in 1969-70 and 2006-07. PTSD identified in 2006-07 was modelled as a latent variable with three manifest indicators (re-experiencing, hyper-arousal and avoidance). Categorical variables were determined by frequency tables for respective group participants. Group differences in continuous variables were analysed by t-test or the Wilcoxon signed rank sum test accounting for non-normal distributions. Categorical variables, chi-square tests or Fisher's Exact Tests were performed when assumptions of chi-square tests were violated. Research participants from 1969-70 and 2006-07 did not indicate a significant difference in demographic, categorical or continuous variables. Initial 1969-70 battlefield psychiatric diagnosis TSD did indicate of a causal link to delayed onset PTSD in research participants in 2006-07. The PTSD (2006-07 diagnosis) indicated a descriptive difference, 64 of the 76 Control met the diagnostic criteria, while 19 of the 21 Experimental met the criteria. A significant difference was identified in the 2006-07 presence and severity of depression, two symptoms (intrusion and avoidance) of PTSD and the reported combat exposure. The prevalence of delayed onset PTSD was also highlighted. Obtaining original battlefield psychiatric diagnoses is rare. Comparison with an identifiable Control group after 35 years informs knowledge of how military personnel cope with battlefield exposure. Specifically concluding that; battlefield exposures during 1969-70 for the majority of the research participants have impacted detrimentally on their psychological and physical health, relationships, employment and ongoing overall wellbeing to this day. Delayed onset PTSD is the principal indicator of this current state for these veterans. / Thesis (Ph.D.) - University of Adelaide, School of Population Health and Clinical Practice, 2008

The primary purposes of this dissertation were to explore relationship between cell free DNA (cf-DNA), creatine kinase (CK), C-reactive protein (CRP), vertical jump testing delayed onset muscle soreness (DOMS) in response to a high-volume resistance training protocol, and to assess the sensitivity of cf-DNA to different resistance training volume loads. The secondary purpose was to examine the relationship between cf-DNA and relative strength. Study 1 was an exploratory attempt to discover relationships between cf-DNA, CK, CRP, delayed onset muscle soreness, and performance variables. Seventeen resistance trained males were recruited, 9 were randomly assigned to receive BCAAs while 8 received a placebo. Participants performed a high-volume resistance training session consisting of the back squat and bench press. Blood was drawn to measure serum cf-DNA, CK, and CRP levels prior to the training session, with cf-DNA collected immediately post, and CK and CRP at 24hr and 48hrs post. Self-reported DOMS on a scale of 1 to 10 was collected prior to training on day 2, day 3, and day 4. SJH, CMJH, and BOSCO were collected on day 1, day 3, and day 4. Fifty-seven correlations were run to explore the relationships between variables. Only the correlation between %Δ DOMS 48hr and %Δ CRP 48hr in the non-supplement group was significant (p = 0.02). The second study, designed to assess the sensitivity of cf-DNA to different resistance training volume loads, consisted of a high-volume resistance training protocol. Blood was drawn immediately before the resistance training session (T1), immediately after the third lifting set (T2), and immediately after the sixth lifting set (T3). cf-DNA increased significantly from T1 to T2 (p < 0.01) and T1 to T3 (p < 0.01). The linear regression model used to examine the capabilities of relative strength to predict %Δ cf-DNA from T1 to T3 was significant (p = 0.04). The results of this study demonstrate the short response time of cf-DNA in relation to variations in resistance training volume-load, suggesting it may be a valuable marker in monitoring the immune response to volume-load. Results also demonstrated the positive relationship between relative strength and %Δ cf-DNA.

Träningsvärk efter fysisk aktivitet är ett välkänt fenomen. Vad som ligger bakomträningsvärkens effekter såsom smärta och nedsatt rörlighet är mindre välkänt. Träningsvärk, eller ”delayed onset muscle soreness” (DOMS), har i forskningenantagits bero på muskelskada och inflammation i skelettmusklerna efter att ovana ellerexcentriska övningar utförts. Försöken att lindra träningsvärkens effekter har varitmånga, t ex genom antiinflammatoriska läkemedel (NSAID). Syftet med dennasystematiska litteraturstudie var att svara på frågeställningen hur ibuprofen påverkarträningsvärk där hypotesen att ibuprofen skulle ha en dämpande effekt på träningsvärkantogs. Nio artiklar granskades för att besvara hypotesen utan att några slutsatser omibuprofens inverkan på träningsvärk kunde dras då resultaten pekade åt olika håll ochinga tendenser kunde observeras. Den slutsats som kunde dras utifrån litteraturstudienvar att ytterligare forskning på området är behövlig. Om studier inte kan visa att NSAIDdämpar träningsvärk så kan det ifrågasättas om inflammation är orsaken tillträningsvärken. Ytterligare studier på området är viktigt då NSAID är vanligtförekommande som smärtlindrande läkemedel samtidigt som det har biverkningar. Det finns också forskning som tyder på att NSAID kan ha negativ inverkan på de positivaeffekter som är av intresse i träningssammanhang.

Massage is often recommended to athletes to facilitate recovery and attenuate DOMS. The purpose of this study was to investigate the effects of massage on perceived muscle soreness and pain, inflammatory and immune markers, ROM, and mood state. Fourteen, recreationally active, women participated in a randomized crossover design study, consisting of 1) 60 min. full body massage following unaccustomed exercise and 2) 60 min. of rest. following unaccustomed exercise. Perceived muscle soreness and pain, active range of motion (ROM), mood state, along with blood concentrations of interleukin-6 (IL-6), C-reactive protein (CRP), creatine kinase (CK), and neutrophil count (NC), was assessed at baseline, 4hrs, and 24hrs following both treatment and control conditions. The aims of this study were 1) to decrease the effects of delayed onset muscle soreness (DOMS), and increase time to recover, and 2) to investigate the effect of massage vs. passive rest on inflammatory and immune markers within the blood. We hypothesized 1) an increase in ROM, a decrease in perceived physical soreness and perceived physical pain, as a result of the massage, compared to control, and 2) a decrease in blood plasma inflammatory markers, CRP, NC, CK, and IL-6, as a result of the massage, compared to control. We found massage following exercise to 1) significantly decreased perceived pain (p=0.001), 2) significantly increased immune iv markers (WBC (p=0.012) and NC (p=0.012)), and 3) significantly decreased ROM (p=0.02), compared to control. Massage had no impact on inflammatory markers (IL-6, CRP, and CK), or mood.

Bien qu’il soit recommandé de pratiquer tout au long de sa vie une activité physique régulière pour se maintenir en bonne santé, peu d’études portent sur les modifications des patrons locomoteurs lors de la répétition de séances à 2 ou 3 jours d’intervalle. Lorsqu’elles sont intenses ou inhabituelles, les formes naturelles de locomotion pédestre – dites de type cycle étirement-détente (CED) – se caractérisent par des perturbations structuro-fonctionnelles et proprioceptives qui peuvent perdurer plusieurs jours. Ces déficiences sont autant de sources de risques accrus de blessures lors de la répétition d’une pratique sportive. Ce travail doctoral a pour objectif (i) d’examiner les effets immédiats et retardés d’exercices épuisants de type CED sur les paramètres neuromécaniques de la locomotion humaine en conditions de marche, de course et de sauts et (ii) d’étudier l’influence combinée d’une pratique pieds nus vs. pieds chaussés. Nos résultats ne révèlent pas de modification significative des patrons locomoteurs en phase de récupération immédiate (post-exercice) mais démontrent l’intervention de stratégies compensatrices et/ou protectrices en phase de récupération retardée (2ème jour posteffort). Ces stratégies diffèrent entre les conditions de marche et de course. Nos travaux soulignent également l’importance de discriminer les stratégies adoptées dès la première minute de l’exercice des ajustements ultérieurs apparaissant pendant la phase dite d’optimisation du CED. Par contre, les différences observées entre les conditions de course pieds nus vs. pieds chaussés restent étonnamment similaires avec la fatigue. Le port de chaussures se traduit par une réduction des chocs d’impact mais également par une restriction des mouvements naturels du pied et par une éversion accrue dont il conviendrait d’étudier les conséquences lors de la répétition en état de fatigue d’exercices de plus longue durée. / Although lifelong practice of moderate amounts of regular physical activity is recommended to ensure a healthy living state, little is know about the modifications of the locomotion patterns when repeating exercises every 2 to 3 days. Intense or unaccustomed stretch-shortening cycle (SSC) forms of ground locomotion are characterized, however, by structuro-functional and proprioceptive impairments that may last for a few days. These impairments may then be expected to increase the risk of injury. The present work aimed (i) to examine the acute and delayed SSC fatigue effects on the neuro-mechanical gait characteristics in walking, running and jumping conditions and (ii) to study the combined influence of a barefoot vs. shod gait condition. Our results did not reveal any modification of the locomotion patterns in the acute recovery phase (post-exercise), but demonstrated compensatory and/or protective strategies in the delayed phase (2 days post-exercise). The observed strategies differed significantly in walking and running. Our results emphasized also the need to differentiate the strategies occurring within the first minute of exercise from the subsequent adjustments related to the progressive SSC pattern optimization. Surprisingly, the observed differences between the barefoot and shod running conditions remained quite independent of the fatigue state. The shod running condition was thus found to attenuate ground impact transmission, but it resulted also in restricted natural foot motions and increased eversion that should be worth re-examining when repeating exercises of longer duration.