Syftet med denna studie var att undersöka samband mellan stress, sömn och skärmtid på smarttelefon. Urvalet bestod av totalt 81 personer på ett universitet i södra Sverige. Två hypoteser testades. 1: hög grad av faktisk skärmtid är relaterat till en hög subjektiv stressupplevelse och hög grad av sömnbesvär. 2: Låg grad av stress är associerat med låg grad av sömnbesvär. Vid insamlingen av data användes, Perceived stress scale 10 (PSS-10) och Karolinska sleep questionnaire (KSQ). För att mäta skärmtid designades två frågor, en med syfte att mäta individers subjektiva upplevelse av mängden skärmtid och en för att mäta den faktiska mängden som kan utläsas via en funktion på individers smarttelefon. Resultatet för hypotes 1 visade att faktisk skärmtid varken samvarierade med stress eller sömnbesvär. Vidare jämfördes också gruppen hög faktisk skärmtid som översteg 29 h/vecka med stress och sömnbesvär. Resultatet indikerade att en hög faktisk skärmtid och stress inte korrelerade och hög faktisk skärmtid och sömnbesvär inte korrelerade. Resultatet för hypotes 2 visade en negativ signifikant korrelation mellan variablerna stress och sömnbesvär. Det innebar att höga värden på total KSQ, mindre sömnbesvär, korrelerar med låga värden på PSS-10, låg nivå av upplevd stress. Sammanfattningsvis visade studiens resultat att faktisk skärmtid inte samvarierade med varken stress eller sömnbesvär. Dock återfanns en korrelation mellan variablerna stress och sömnbesvär. / The purpose of this study was to investigate the relationship between stress, sleep and screen time on smartphones. The sample consisted of a total of 81 people at a university in southern Sweden. Two hypotheses were tested. 1: High degree of actual screen time is related to a high subjective stress experience and high degree of sleep disturbance. 2: Low levels of stress are associated with low levels of sleep disturbance. In the data collection, Perceived stress scale 10 (PSS-10) and Karolinska sleep questionnaire (KSQ) were used. To measure screen time, two questions were designed, one aimed at measuring individual's subjective experience of the amount of screen time and one to measure the actual amount that can be read through a function on an individual's smartphone. The result for Hypothesis 1 showed that actual screen time did not co-vary with stress or sleep disturbance. Furthermore, the group, high actual screen time exceeding 29 h / week was also compared with the variables stress and sleep disturbance. The result indicated that a high actual screen time and stress did not correlate, and high actual screen time and sleep disturbances did not correlate either. The result for Hypothesis 2 showed a negatively significant correlation between the variables stress and sleep disorders. This meant that high values of total KSQ, less sleep disturbances, correlated with low values of PSS-10, low levels of stress. In summary, the study's results showed that actual screen time did not correlate with neither stress nor sleep disturbance. However, a correlation was found between the variables stress and sleep disturbance.

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Loneliness occurs in the absence of belonging or social connectedness and has been linked to many physical and mental health problems. Among these conditions are depression, anxiety, sleep disturbance, and stress. College students report these four conditions as the largest barriers to good academic performance. For as much is known about loneliness, much less is known about belonging and health or the role loneliness plays in these relationships prompting a need for investigation. Using a sample of 301 university students, we replicated previous findings that loneliness predicts depression, anxiety, sleep disturbance, and stress. Next we replicated and contributed new findings for the relationship between social connectedness and the same health outcomes of interest. Previous research has found gender to be a moderator in the relationship between loneliness and social connectedness. The current study found no evidence of moderation. Based upon the available literature, it was hypothesized that loneliness would mediate the relationship between social connectedness and the health outcomes of interest. Using conditional process modeling, loneliness was found to be a mediator in every case. These findings validate previous findings on the effects of loneliness on health. They also highlight the significance of social connectedness as a factor in health. Future research should investigate the effectiveness of social connectedness as focal point for treatment of mental and physical health conditions.

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研究目的：在現今高壓力與忙碌的社會中，失眠是很常見的問題。在國外的失眠的相關調查結果顯示，失眠的盛行率會受對於失眠定義之嚴謹程度影響，而有大範圍的變異，其範圍約落在6~48%。當定義符合臨床上之診斷標準時，失眠盛行率約降至6~15%。因此，受失眠之苦的個體中，有一大部分可能未達診斷標準，而為暫時性或急性失眠之患者。然而過去的研究大多將重點聚焦在符合失眠疾患診斷的失眠族群上，針對剩下雖未符合失眠疾患診斷標準，但又飽受失眠症狀困擾之群體，卻是缺乏探討。此外，失眠的縱貫研究中也發現，暫時性失眠個體到最後會有一部份會發展成為慢性失眠。因此，針對這群為數不少，且容易經歷暫時性失眠之個體，若能更加了解其暫時性失眠發生的成因與相關機制，便能及早介入，協助個體不落入慢性失眠的惡性循環中。過去研究發現，暫時性失眠的發生，最常見的促發因素為壓力事件。Drake、Richardson、Roehrs、Scofield與Roth（2004）便發展出福特壓力失眠反應量表（Ford Insomnia Response to Stress Test, FIRST）來測量個體特質上遭遇壓力情境的睡眠脆弱程度；且研究結果也發現個體身心激發程度是和睡眠脆弱特質息息相關的因素之一。由於過去在失眠的相關研究中，少有研究探討暫時性失眠的相關機制，因此，本研究從和暫時性失眠相關的壓力下睡眠脆弱特質出發，探討此脆弱特質與過度激發的關聯性，並欲探討其他會提升激發程度的相關認知與情緒因子，如：失功能信念、情緒反應性、反芻特質、情緒遲惰特質和此特質間的關係。本研究假設個體對於睡眠的失功能信念與對負向事件的情緒反應性可能會透過提高睡前身心的激發程度，而使個體容易經歷壓力下的急性失眠，增加睡眠的脆弱性；即睡前身心激發程度在睡眠的失功能信念與情緒反應性對睡眠脆弱性的關係間扮演一中介作用之角色。另外，也假設睡眠的失功能信念與情緒反應性與睡前身心激發程度間的關係，會分別受反芻特質與情緒遲惰特質調節，即具有高反芻特質與高情緒遲惰特質者，其睡眠的失功能信念與情緒反應性與睡前身心激發程度間的關係更強烈，因此在壓力下，其有較高的睡眠脆弱性，容易經歷暫時性失眠。 研究方法：本研究於大學中招募60位20~35歲之受試者（男：22位；女：38位），經晤談以確認受試者符合收案標準後，將請受試者填寫研究相關問卷（包含：壓力下失眠反應量表、反芻型反應風格短版量表、睡前激發程度量表、睡眠失功能信念及態度簡式量表、情緒反應量表、失眠嚴重度量表、貝克憂鬱量表第二版與貝克焦慮量表）；並在詳細解釋情緒經驗取樣流程後，請受試者回到日常生活環境中進行連續三天的情緒遲惰經驗取樣紀錄。 研究結果：根據執行檢驗中介效果步驟的階層迴歸與Sobel test的分析結果，睡前認知激發程度對擔憂的失功能信念與情緒持續性和睡眠脆弱性間的關係具中介的作用；即對睡眠有過度擔憂的失功能信念、情緒持續性較久皆可能會提高睡前認知激發程度而增加壓力下的睡眠脆弱性。其他失功能信念之向度，如：知覺失眠造成的影響、對睡眠的期待、藥物使用，以及情緒反應性之其他向度，如：情緒敏感度與情緒激發度對睡前認知過度激發狀態無顯著的預測力。另外，根據執行檢驗調節效果步驟的階層迴歸分析結果，反芻特質與情緒遲惰特質分別在失功能信念與情緒反應性對認知激發程度的關係上，皆未有顯著的調節效果。 研究結論：研究結果部分支持身心激發程度為對睡眠的失功能信念與情緒反應性影響壓力下睡眠脆弱性的中介因子之假設。本研究發現過度擔憂睡眠的信念以及情緒持續度較久這兩個因子會分別獨立地提高睡前認知激發程度，顯示睡前認知激發程度分別受認知與情緒因素影響；且相較與睡前的生理激發狀態，睡前的認知激發對於壓力下的失眠反應之影響具有顯著的預測力，顯示認知激發在失眠的前置因子中可能扮演較重要的角色。另外，反芻特質與情緒遲惰特質分別在失功能信念與情緒反應性對認知激發程度的關係上，皆未有顯著的調節效果，此部份不符合研究預期。基於本研究結果，在臨床上對於容易經歷失眠之個體，若能及早調整對於睡眠的擔憂相關的信念，並學習有效調節情緒的方式，皆可有效降低個體睡前的認知激發活動，減少失眠的發生率與改善失眠症狀，避免使其落入失眠慢性化的惡性循環中。